What “hidden fat” means for metabolism

What “hidden fat” means for metabolism

Metabolism isn’t just about calories in and calories out. It’s about where your body stores fat, how much it can store safely, and what happens when that system gets overwhelmed. Recent research is transforming our understanding of metabolic health, and one of the most revolutionary insights is this: everyone has a Personal Fat Threshold. And it’s not always visible from the outside.

It’s important to note that metabolic health isn’t defined by appearance. This isn’t about chasing a certain body size. It’s about understanding how your body functions and making informed, personalized choices to support long-term health.

This concept, along with the Twin Cycle Hypothesis, has dramatically shifted how we understand conditions like insulin resistance, prediabetes, and type 2 diabetes. Together, they offer a new framework for prevention and early intervention—even in people with a “normal” weight.

What is the personal fat threshold?

The Personal Fat Threshold refers to the idea that each person has a unique capacity to store fat in subcutaneous tissue (the fat under the skin). Once that threshold is exceeded, fat begins accumulating in places it shouldn’t, such as the liver, pancreas, and around internal organs. This type of fat is called ectopic fat, and it’s strongly linked to insulin resistance and metabolic dysfunction.
Your threshold is influenced by genetics, early-life environment, hormones, and lifestyle. Some people can store more fat under the skin without issue, while others start storing ectopic fat with very little weight gain.

This explains why metabolic issues can develop even in individuals who are not visibly overweight. It’s also a major reason why BMI often fails to capture true metabolic risk.

Signs you may have exceeded your personal fat threshold

You don’t need an MRI to get insight into ectopic fat levels. Several simple clinical markers and tools can help determine whether someone may have surpassed their threshold:
  • Waist-to-height ratio > 0.5
  • Elevated fasting insulin, glucose, or HOMA-IR
  • Elevated liver enzymes (AST/ALT), triglycerides, or signs of fatty liver
  • FibroScan® results showing increased liver stiffness or fat accumulation
  • InBody or DEXA scan showing increased visceral fat
If these markers are trending upward, your body is likely storing fat where it shouldn't. That can signal growing metabolic risk, regardless of your weight.

The Twin Cycle Hypothesis: How fat drives dysfunction

Once fat spills over into the liver, insulin resistance develops. The pancreas compensates by producing more insulin, which leads to more fat being made and stored, particularly in the liver and eventually the pancreas. This creates a vicious cycle of worsening insulin resistance and beta-cell dysfunction.

This “twin cycle” can quietly build over years until blood sugar finally rises and a diagnosis like prediabetes or type 2 diabetes is made. But here’s the good news: early intervention can interrupt the cycle and even reverse the damage.

6 targeted approaches to reverse ectopic fat and reclaim your metabolism

These interventions are not just about weight loss. They’re about improving where fat is stored and how your metabolism functions:

1. Create a Calorie Deficit: Research shows that a  modest reduction in weight (as little as 6.5% for those at a normal BMI and up to 15% for those who are overweight or obese) can significantly reduce fat in the liver and pancreas and improve insulin sensitivity.

2. Reduce Saturated Fat Intake: Lowering intake of saturated fats, particularly from processed foods and certain animal products, has been shown to decrease liver fat and support healthier insulin response.

3. Engage in Regular Physical Activity: Both aerobic and resistance training increase fat oxidation, reduce liver fat, and improve insulin signaling, especially when done consistently.

4. Prioritize Quality Sleep: Chronic sleep deprivation disrupts insulin and appetite-regulating hormones. Aim for 7–9 hours of high-quality sleep to support metabolic regulation.

5. Manage Stress Effectively: Elevated cortisol promotes visceral fat storage and reduces subcutaneous fat buffering. This shifts fat toward organs and increases metabolic risk. Stress management strategies like mindfulness, breathwork, and movement can blunt this effect.

6. Optimize Hormonal Health:Thyroid imbalances, insulin resistance, and sex hormone fluctuations can all impair metabolic function. Comprehensive hormone testing and individualized support can help restore balance and improve fat distribution.

Final thoughts

The idea that your body has a personal limit for safely storing fat—and that exceeding it may be the real trigger for metabolic breakdown—is a profound shift. It explains why conventional weight-focused metrics miss the mark and why personalized interventions matter.
Understanding your personal fat threshold is one of the most powerful steps you can take to protect your metabolic health, prevent disease, and optimize longevity.

Ready to take the next step?

Book a visit with a BioLounge clinical nutritionist to dive deeper into precision nutrition strategies tailored to your unique metabolism. We’ll help you understand your body’s signals, optimize your nutrition, and create a personalized plan to support lasting metabolic health.

References:
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  • Taylor R. Type 2 diabetes: Etiology and reversibility. Diabetes Care. 2013;36(4):1047–1055. doi:10.2337/dc12-1805

  • Lim EL, Hollingsworth KG, Aribisala BS, Chen MJ, Mathers JC, Taylor R. Reversal of type 2 diabetes: Normalisation of beta cell function in association with decreased pancreas and liver triacylglycerol. Diabetologia. 2011;54(10):2506–2514. doi:10.1007/s00125-011-2204-7

  • Taylor R, Calorie restriction for long-term remission of type 2 diabetes. Clin Med (Lond). 2019;19(5):367–372. doi:10.7861/clinmedicine.19-5-367

  • Taylor R, Holman RR. Normal weight individuals who develop type 2 diabetes: the personal fat threshold. Clin Sci (Lond). 2015;128(7):405–410. doi:10.1042/CS20140268